Amyloid beta (Aβ) protein is a key factor in the development of Alzheimer's disease and other types of dementia. It is a naturally occurring protein that is usually broken down and removed from the brain under normal conditions. However, in Alzheimer's disease, Aβ builds up and forms plaques between nerve cells. These plaques interfere with neuron communication, causing cell death and cognitive decline. While Aβ plays a central role in Alzheimer's, other factors, including inflammation and neurofibrillary tangles, also contribute to the progression of the disease.
Track 12-1: Amyloid Beta (Aβ) Production and Clearance
Track 12-2: Aβ Aggregation and Plaque Formation
Track 12-3: Aβ-Induced Neurotoxicity
Track 12-4: Aβ and Tau Protein Interactions
Track 12-5: Aβ as a Therapeutic Target
All accepted abstracts will be published in respective Allied Academies Journals.
Abstracts will be provided with Digital Object Identifier by
